A Biomechanical and Therapeutic Review of Equine Laminitis

Two-Part EVA Clog System with Advanced Manufacturing Applications

Author: Mark Caldwell PhD FWCF.

Email: info@hoofflix.com


Keywords: equine laminitis; biomechanics; EVA clog; adhesive farriery; 3D printing; P3 displacement; hoof rehabilitation


Abstract

Equine laminitis is a multifactorial, biomechanically driven disease characterized by inflammation and failure of the dermal-epidermal lamellar interface, leading to displacement of the distal phalanx (P3) and significant lameness. Mechanical forces—ground reaction forces (GRF) and tension from the deep digital flexor tendon (DDFT)—compound lamellar failure, resulting in P3 rotation or sinking. This comprehensive review synthesizes current understanding of etiologies (endocrinopathic, inflammatory, mechanical) and evaluates farriery methods through a SWOT analytical lens. We introduce a modular, two-part glue-on EVA clog system featuring a removable treatment plate and adhesive base, designed to redistribute load, stabilize the hoof capsule, and avoid nail trauma. A case is made for integrating 3D scanning and additive manufacturing to custom-produce clogs optimized for individual hoof mechanics. Future research directions include biomechanical trials, outcome tracking, and wider clinical evaluation.


1. Introduction

Laminitis in horses involves failure of the laminar interface suspending the distal phalanx (P3) within the hoof capsule. While inflammation has long been a focus, accumulating evidence indicates that laminitis is predominantly a biomechanical failure, triggered by metabolic, inflammatory, or structural insults and exacerbated by mechanical load (Mitchell, Fugler & Eades, 2014; Pollitt & Visser, 2010). Radiographic markers such as lamellar zone widening, palmar angle shift, and P3 displacement correlate strongly with clinical severity (Redden, 2003). Catastrophic “sinkers” exhibit vertical droop of P3 with concomitant proximal hoof capsule movement, a pattern consistent with Newtonian prediction of force vectors and tissue yield thresholds (Coffman et al., 1970). Current farriery lacks consistency and often involves invasive shoeing techniques that risk further damage. This work proposes a trauma-free, modular, EVA-based clog system that addresses biomechanical deficits, and examines opportunities for individualised production via 3D printing.

Figure 1. Diagram illustrating biomechanical forces in laminitis: GRF vector, DDFT tension, lamellar interface, and the mechanics of P3 rotation/sinking.

2. Pathophysiology and Etiology

Laminitis originates from systemic triggers that weaken the basement membrane and lamellar suspensory apparatus. Etiologies include endocrinopathy (EMS, PPID), sepsis, carbohydrate overload, and support-limb compromise (Pollitt & Visser, 2010). Once structural integrity is lost, GRF and DDFT forces drive P3 into pathological positions, either rotating dorsally (rotation) or descending vertically through the weakened lamellar bed, manifesting as “sinker” syndrome (Coffman et al., 1970).

Radiographic thresholds inform prognosis and management strategies. Lamellar zone widths over 18 mm, palmar angles exceeding 11°, or P3 rotations over 10–12° are strongly associated with severe outcomes (Redden, 2003; Orsini et al., 2010).

Figure 2. Radiographic series showing progressive stages: (a) healthy hoof; (b) rotation-phase; (c) sinking-phase laminitis. Measurement annotations detail lamellar zone, palmar angle, and P3 alignment (photo credit; The Laminitis Site).

Table 1. Radiographic criteria for laminitis severity

ParameterNormalPathologic
Lamellar zone width15–17 mm>18 mm
Palmar angle3–5°>11°
P3 rotation<5°>10–12°

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